Category: Aging research

Recent research published in *Nature Aging* has identified a single protein, FTL1, as a potential driver of age-related cognitive decline in mice. This groundbreaking discovery has led many to question whether this protein is present in foods and if diet can influence its levels. However, FTL1 is not a protein consumed in the diet; it is produced within the body, specifically in the brain.

While both Urolithin A and NAD+ are recognized for their roles in supporting cellular health and influencing the aging process, they function through distinct biological mechanisms. Urolithin A is a postbiotic compound associated with promoting mitochondrial recycling, while NAD+ is a fundamental coenzyme vital for numerous metabolic pathways.

Early animal studies show that senolytic drugs can extend lifespan and improve healthspan, suggesting their potential to treat age-related diseases. However, these compounds are not suitable for everyone, making it crucial to understand who should not take senolytics due to potential risks and significant safety concerns.