Tag: Thiomolybdate

While excess molybdenum has been definitively proven to cause copper deficiency in ruminant animals like cattle and sheep, the interaction is not considered significant in humans at typical dietary intake levels. It is a crucial topic for understanding mineral metabolism, especially for those concerned about nutrient balance or managing certain health conditions.

Excessive dietary intake of molybdenum is documented to produce severe copper deficiency in ruminant animals, a phenomenon observed worldwide known as molybdenosis or "teart". This potent mineral antagonism is a complex biochemical process with profound implications for both animal and, under specific conditions, human health, affecting how the body absorbs and utilizes these vital trace minerals.